首站-论文投稿智能助手
典型文献
Fetuin-A is an immunomodulator and a potential therapeutic option in BMP4-dependent heterotopic ossification and associated bone mass loss
文献摘要:
INTRODUCTION Heterotopic ossification (HO) is a potentially severe pathologic process defined as the pathological growth of extraskeletal bone.1–3 HO can be acquired or hereditary.
文献关键词:
作者姓名:
Chen Kan;Jiazhao Yang;Haitao Fan;Yuanjuan Dai;Xingxing Wang;Rui Chen;Jia Liu;Xiangyue Meng;Wei Wang;Guiling Li;Jiao Zhou;Ya Zhang;Wanbo Zhu;Shiyuan Fang;Haiming Wei;Hong Zheng;Siying Wang;Fang Ni
作者机构:
Department of Pathophysiology,School of Basic Medical Sciences,Anhui Medical University,Hefei,China;Department of Hematology,The First Affiliated Hospital of USTC,The CAS Key Laboratory of Innate Immunity and Chronic Disease,School of Basic Medical Sciences,Division of Life Sciences and Medicine,University of Science and Technology of China,Hefei,China;Institute of Immunology,University of Science and Technology of China,Hefei,China;Department of Orthopaedics,The First Affiliated Hospital of USTC,Hefei,China;Department of Orthopaedics,Fuyang Hospital of Anhui Medical University,Fuyang,China
引用格式:
[1]Chen Kan;Jiazhao Yang;Haitao Fan;Yuanjuan Dai;Xingxing Wang;Rui Chen;Jia Liu;Xiangyue Meng;Wei Wang;Guiling Li;Jiao Zhou;Ya Zhang;Wanbo Zhu;Shiyuan Fang;Haiming Wei;Hong Zheng;Siying Wang;Fang Ni-.Fetuin-A is an immunomodulator and a potential therapeutic option in BMP4-dependent heterotopic ossification and associated bone mass loss)[J].骨研究(英文版),2022(04):867-881
A类:
Heterotopic
B类:
Fetuin,is,immunomodulator,therapeutic,option,BMP4,dependent,heterotopic,ossification,associated,bone,mass,loss,INTRODUCTION,HO,potentially,severe,process,defined,pathological,growth,extraskeletal,can,be,acquired,hereditary
AB值:
0.694132
相似文献
Molecular pathogenesis of acetaminophen-induced liver injury and its treatment options
Xiaopeng CAI;Huiqiang CAI;Jing WANG;Qin YANG;Jun GUAN;Jingwen DENG;Zhi CHEN-State Key Laboratory for Diagnosis and Treatment of Infectious Diseases,National Clinical Research Center for Infectious Diseases,Collaborative Innovation Center for Diagnosis and Treatment of Infectious Diseases,the First Affiliated Hospital,Zhejiang University School of Medicine,Hangzhou 310003,China;Department of Clinical Medicine,University of Aarhus,Palle Juul-Jensens Boulevard 82,8200 Aarhus N,Denmark;Department of Pathology,Key Laboratory of Disease Proteomics of Zhejiang Province,Zhejiang University School of Medicine,Hangzhou 310058,China;Department of Pathology,Women’s Hospital,Zhejiang University School of Medicine,Hangzhou 310058,China
FGF2 is overexpressed in asthma and promotes airway inflammation through the FGFR/MAPK/NF-κB pathway in airway epithelial cells
Yuan-Yang Tan;Hui-Qin Zhou;Yu-Jing Lin;Liu-Tong Yi;Zhuang-Gui Chen;Qing-Dong Cao;Yan-Rong Guo;Zhao-Ni Wang;Shou-Deng Chen;Yang Li;De-Yun Wang;Yong-Kang Qiao;Yan Yan-Guangdong Provincial Key Laboratory of Biomedical Imaging and Guangdong Provincial Engineering Research Center,the Fifth Affiliated Hospital,Sun Yat-Sen University,Zhuhai 519000,Guangdong,China;Department of Pathology,the Fifth Affiliated Hospital,Sun Yat-Sen University,Zhuhai 519000,Guangdong,China;Department of Pediatrics,the Third Affiliated Hospital,Sun Yat-Sen University,Guangzhou 510630,China;Department of Cardiothoracic Surgery,the Fifth Affiliated Hospital,Sun Yat-Sen University,Zhuhai 519000,Guangdong,China;Department of Otolaryngology,Yong Loo Lin School of Medicine,National University Health System,National University of Singapore,Singapore 119228,Singapore;BGI-Shenzhen,Shenzhen 518083,Guangdong,China;Central Laboratory,the Fifth Affiliated Hospital,Sun Yat-Sen University,Zhuhai 519000,Guangdong,China
Deficient Rnf43 potentiates hyperactive Kras-mediated pancreatic preneoplasia initiation and malignant transformation
Xian Zhou;Zhichao Sun;Mengdi Zhang;Xiaoyu Qu;Shuhui Yang;Lianmei Wang;Yanling Jing;Li Li;Weiwei Deng;Fangming Liu;Jin Di;Jie Chen;Jian Wu;Hongbing Zhang-State Key Laboratory of Medical Molecular Biology,Department of Physiology,Institute of Basic Medical Sciences and School of Basic Medicine, Chinese Academy of Medical Sciences and Peking Union Medical College,Beijing, China;Institute of Cancer Stem Cell,Dalian Medical University,Dalian,China;Institute of Chinese Materia Medica, China Academy of Chinese Medical Sciences,Beijing,China;Department of Pathology,Peking Union Medical College Hospital,Chinese Academy of Medical Sciences,Beijing, China;MyGenostics Inc.,Beijing,China
Treatment of autosomal recessive hearing loss via in vivo CRISPR/Cas9-mediated optimized homology-directed repair in mice
Xi Gu;Xinde Hu;Daqi Wang;Zhijiao Xu;Fang Wang;Di Li;Geng-lin Li;Hui Yang;Huawei Li;Erwei Zuo;Yilai Shu-ENT institute and Department of Otorhinolaryngology,Eye & ENT Hospital,State Key Laboratory of Medical Neurobiology and MOE Frontiers Center for Brain Science,Fudan University,Shanghai,China;Institutes of Biomedical Sciences,Fudan University,Shanghai,China;Department of Otolaryngology,the First Affiliated Hospital of Fujian Medical University,Fuzhou,China;NHC Key Laboratory of Hearing Medicine(Fudan University),Shanghai,China;Institute of Neuroscience,State Key Laboratory of Neuroscience,Key Laboratory of Primate Neurobiology,CAS Center for Excellence in Brain Science and Intelligence Technology,Shanghai Research Center for Brain Science and Brain-Inspired Intelligence,Shanghai Institutes for Biological Sciences,Chinese Academy of Sciences,Shanghai,China;State Key Lab for Conservation and Utilization of Subtropical Agric-Biological Resources,Guangxi University,Nanning,China;Shenzhen Branch,Guangdong Laboratory for Lingnan Modern Agriculture,Genome Analysis Laboratory of the Ministry of Agriculture,Agricultural Genomics Institute at Shenzhen,Chinese Academy of Agricultural Sciences,Shenzhen,China;The Institutes of Brain Science and the Collaborative Innovation Center for Brain Science,Fudan University,Shanghai,China
机标中图分类号,由域田数据科技根据网络公开资料自动分析生成,仅供学习研究参考。